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endothelial dysfunction

Vertaling van "endothelial dysfunction" in Chinees

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内皮功能障碍
内皮功能紊乱
皮功能障碍
Although other factors, such as diet may contribute to these findings, physical activity is known to reduce systemic inflammation and endothelial dysfunction, both hypothesized to have a role in the pathogenesis of age-related macular degeneration.
尽管节食等因素对这些结果可能有影响,但有人根据体育活动已知可以减轻全身炎症和内皮功能障碍,推测其对于年龄相关黄斑变性的发病也有作用。
These results indicate that endothelial gap junctions are highly involved in atherogenesis and suggest that down-regulation of endothelial gap junction may be used as a marker of endothelial dysfunction.
这些结果说明内皮缝隙连接与动脉粥样硬化的发生高度相关,提示我们可以把内皮缝隙连接下调作为内皮功能障碍的一个标志。
This article should summarize the present state concerning smooth muscle contraction, endothelial dysfunction, inflammatory changes, gene expression, in the genesis of vasospasm following aneurysmal subarachnoid hemorrhage.
本文将总结现在关于平滑肌收缩、内皮功能紊乱、炎性改变和基因表达导致动脉瘤性蛛网膜下腔出血后血管痉挛的阐述。
Reactive oxygen species can mediate signalling pathways in cardiac hypertrophy and cardiomyocyte apoptosis, and lead to endothelial dysfunction by inactivating nitrogen monoxide.
活性氧通过信号通路介导心肌细胞肥大和凋亡;通过灭活一氧化氮等机制导致内皮功能紊乱。
Oxidative stress in vascular endothelial cells, which line major and minor blood vessels, can lead to endothelial dysfunction and is believed to be an important contributing factor in the development of systemic cardiovascular disease, complications of diabetes, chronic kidney disease and other forms of organ failure, and a number of other aging-related diseases, including degenerative diseases of the central nervous system and the retina.
内衬于大血管和小血管中的血管内皮细胞中的氧化应激可导致内皮功能障碍并且被认为是发生全身性心血管疾病、糖尿病并发症、慢性肾病和其它形式的器官衰竭以及许多其它衰老相关性疾病 ( 包括中枢神经系统和视网膜的变性疾病 ) 的重要促成因素。
This article should summarize the present state concerning smooth muscle contraction, endothelial dysfunction, inflammator
本文将总结现在关于平滑肌收缩、内皮功能紊乱、炎性改变和基因表达导致动脉瘤性蛛网膜下腔出血后血管痉挛的阐述。
It is suggested that the effects of Hirudo on reversion of endothelial dysfunction might be important mechanism of its antiatherosclerotic effect.
上述结果提示水蛭逆转内皮功能障碍可能是其抗动脉粥样硬化作用的重要机制。
Patients with OSA with low cardiovascular risk statushad increased oxidant production in the microcirculation and endothelial dysfunction, both of which improved with treatment.
OSA心血管低风险的病人微循环氧化物升高,内皮功能障碍,这些在治疗后都有改善。
The biomarkers of endothelial dysfunction, serum endothelin-1, E-Selectin and MMP-9, levels were also lower for the patients taking vitamin D.
服用维生素D的患者,内皮功能障碍的生物标志物如:血清内皮素1、E -选择和MMP-9,水平也较低。
Our study shows that serum PTX3 levels are associated with endothelial dysfunction in patients with stage 1 diabetic CKD, independent of CRP.
我们的研究表明在糖尿病肾病1期患者中血清PTX3水平与内皮功能障碍相关,与CRP无关。
LOX-1 was identified from endothelial cells as the main receptor specially recognizing and uptaking the ox-LDL, LOX-1 plays a key role in the endothelial dysfunction triggered by ox-LDL.
凝集素样氧化低密度脂蛋白受体(LOX-1)是内皮细胞膜上特异性识别摄取ox-LDL的最主要受体,LOX-1在介导ox-LDL导致内皮功能紊乱的过程中起关键作用。
to endothelial dysfunction, and she in turn to atherosclerosis,
到皮功能障碍,并且她在转到动脉粥样硬化
for endothelial dysfunction in patients with coronary artery disease
对冠状动脉疾病患者是一个内皮功能障碍的危险因素
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